These are from a strong, healthy brain in later life from lower risk of premature death.
But some of us are more likely to build muscle than others. Now a new study is available in Nature Communications magazine, which provides molecular explanations for it.
It also shows in the new study why some people get training in more responsive muscles rather than endurance training and aerobic exercise.
Sarah Lessard, PhD - Massachusetts Boston Joslin Diabetes Center Clinical Research Assistant, Behavior and Research Department Results - The first author of the paper.
In question the molecule is a protein called C-Jun N terminal kinase (JNK). Speaking of which, Lessard said, "It's like a switch [...] switch is on, you have to increase the muscles. You will have the ability to turn off, to adapt your muscles."
He said, "We have identified a biological path for exercise activation which has not been studied."
How does JNK affect muscle growth?
The basis of this new study is the work of the same scientist. In his previous study, Lessard and his colleagues studied the genetic make-up of the breed to create a good or very poor reaction to their endurance endurance exercise.
At that time, the researchers first discovered the molecule is the reason for the expression in rats on a treadmill of the JNK route.
In this new study, researchers hope to know why JNK plays such an important role.
Thus, knock lesser and in the team, rats were compared to a group of JNK genes, and their behavior compared to the behavior of common mice.
Effluent rodents remain healthy and keep moving on the treadmill from time to time.
After this, the researchers trained both groups of rodents. At this point, scientists have found that the aerobic capacity increases JNK-free mice, blood vessels and strength and muscle fibers, thereby enhancing endurance.
On the contrary, in the growth of the muscles muscles, without JNK rodents, their muscles failed to increase traditional mice increased their muscles.
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